marijuana during pregnancy....?

cackpircings

Well-Known Member
It’s not crack! My wife did, my baby girl is SMART (started reading at one year). Beautiful (no weird marks of anything) and funny (don’t know if the weed helped. So do what you think is best for your kid. Don’t let the internet sway you. Of course the drug companies would launch a campaign to say its bed, shit it helps people with cancer so they can eat and not get sick, it would work the same way for a pregnant woman. I found out she was doing it while I was in Iraq, and at first I was like wait! Then I did a whole lot of reading and research, and found that ancient china’s woman used to use it for just that reason (morning sickness, and nausea) I have seen the effects on crack babies (my uncle manufactures special beds for them). That is some shit! They shake and never sleep. That is some sick shit, and let’s not forgets the woman that drink or smoke cigarettes while prego that shit is crazy. My mom (in the 1980) smoked cigarettes and drank and I turned out fine however both my brothers are really slow, and now she is addicted to pills. Points being don’t let the internet sway your opinion. Make your own mind up that’s why you’re a grown ass man having a baby.
 

cackpircings

Well-Known Member
She told me that she didn’t do it for the first month... she did read a lot about it before doing it. There is much worse things you can do to a child and I know it’s not easy being prego! Let the girl make her own decisions.
 

fdd2blk

Well-Known Member
crack is bad. i was trying to point out that if pot was bad you'd know it. there'd be headlines "pot babies are being born everywhere. we need more incubators". i don't see that anywhere.
 

cackpircings

Well-Known Member
I found so many bull shit responses on the internet where clearly people are basing their facts on emotion rather than hard evidence (which we all know always prevails). So trust me if it did hurt the baby they would use that as a reason to make a sensible argument out of why they need to keep weed illegal (which they don’t have one anymore) I’m not saying go out and be a pot head while prego, because too much of anything is bad IE; eating McDonalds everyday supersized is dumber than smoking mass weed everyday because obviously one will kill you the other will just make the idea sound good at the time. So all in all yes I feel it is ok to use it in the morning to cure the sickness, and at night to just relax. To some things up… I read a lot of stories of PEOPLE saying they knew someone that knew someone… so on and so forth… that smoked weed and their kid had an abnormal sized head. This is malarkey, my kid came out and lifted her head right up and is totally proportionate. SO what is the Argument? Is it going to hurt the kid no… Is smoking weed bad for any of us? Not really! We need not take things to the extreme and it will never get out of control. Some things my wife did do: She didn’t eat any fast food at all. She would take her vitamins religiously, and would read to her stomach and my little girl loves to read now so I know it helped. The baby loves music as well and would kick all around when she would pump it up. So none the less, good luck on the boy and keep on keepn on!
 

cackpircings

Well-Known Member
crack is bad. i was trying to point out that if pot was bad you'd know it. there'd be headlines "pot babies are being born everywhere. we need more incubators". i don't see that anywhere.

that is what I am saying FDD.

When I was younger we had all led paint toys on our playgrounds n school. It is good to think of the baby first, but throwing up is a violent response of the body, and the constriction can’t be too good for the baby. I forgot to also say that my baby also would sleep form 6 pm in the evening to 7 am in the morning. That is more than most parents can say for their kid. Was it the weed? I don’t know, but if I ever have a second kid and it works out to be the same as my current, I am going to say that maybe it had something to do with it.
 

stopcallingmedude

Well-Known Member
hey just wondering if anyone has any good links to website with info on smoking/eating marijuana while pregnant? if u have sum links that are good and from a reliable source ild love to see them!... thanks alot RIU!
dirtyboy... u got ur phd where?
i have my phd and i have multiple articles written by colleagues that you can read if you'd like to pm me with an e-mail address. they are rather technical but they are pretty solid. i'll put a few of the abstracts up now:

Fetal development is a period sensitive to environmental influences such as maternal drug use. The most commonly used illicit drug by pregnant women is marijuana. The present study investigated the effects of in utero marijuana exposure on expression levels of opioid-related genes in the human fetal forebrain in light of the strong interaction between the cannabinoid and opioid systems. The study group consisted of 42 midgestation fetuses from saline-induced voluntary abortions. The opioid peptide precursors (preprodynorphin and preproenkephalin (PENK)) and receptor (mu, kappa and delta) mRNA expression were assessed in distinct brain regions. The effect of prenatal cannabis exposure was analyzed by multiple regression controlling for confounding variables (maternal alcohol and cigarette use, fetal age, sex, growth measure and post-mortem interval). Prenatal cannabis exposure was significantly associated with increased mu receptor expression in the amygdala, reduced kappa receptor mRNA in mediodorsal thalamic nucleus and reduced preproenkephalin expression in the caudal putamen. Prenatal alcohol exposure primarily influenced the kappa receptor mRNA with reduced levels in the amygdala, claustrum, putamen and insula cortex. No significant effect of prenatal nicotine exposure could be discerned in the present study group. These results indicate that maternal cannabis and alcohol exposure during pregnancy differentially impair opioid-related genes in distinct brain circuits that may have long-term effects on cognitive and emotional behaviors.

here's another:

The endocannabinoid system, including its receptors (CB1 and CB2), endogenous ligands(‘endocannabinoids’), synthesising and degrading enzymes, as well as transporter molecules, has been detected from the earliest stages of embryonic development and throughout pre- and postnatal development. In addition, the endocannabinoids, notably 2-arachidonyl glycerol, are also present in maternal milk. During three distinct developmental stages (i.e. embryonic implantation, prenatal brain development and postnatal suckling), the endocannabinoid system appears to play an essential role for development and survival. Thus, during early pregnancy, successful embryonic passage through the oviduct and implantation into the uterus both require critical enzymatic control of optimal anandamide levels at the appropriate times and sites. During foetal life, the cannabinoid CB1 receptor plays a major role in brain development, regulating neural progenitor differentiation into neurones and glia and guiding axonal migration and synaptogenesis. Postnatally, CB1 receptor blockade interferes with the initiation of milk suckling in mouse pups, by inducing oral motor weakness, which exposes a critical role for CB1 receptors in the initiation of milk suckling by neonates, possibly by interfering with innervation of the tongue muscles. Manipulating the endocannabinoid system by pre- and ⁄ or postnatal administration of cannabinoids or maternal marijuana consumption, has significant, yet subtle effects on the offspring. Thus, alterations in the dopamine, GABA and endocannabioid systems have been reported while enhanced drug seeking behaviour and impaired executive (prefrontal cortical) function have also been observed. The relatively mild nature of the disruptive effects of prenatal cannabinoids may be understood in the framework of the intricate timing requirements and frequently biphasic effects of the (endo)cannabinoids. In conclusion, the endocannabinoid system plays several key roles in pre- and postnatal development. Future studies should further clarify the mechanisms involved and provide a better understanding of the adverse effects of prenatal exposure, in order to design strategies for the treatment of conditions such as infertility, mental retardation and failure-to-thrive.

you can also just use common sense. during pregnancy the fetus is 100% linked to the mother. anything that goes into the mother is filtered through the ambiotic fluid and into the child. smoking marijuana puts carcinogens into the body... and thus into the baby. hope that helps. :peace:
 

peacemane420

Well-Known Member
i have my phd and i have multiple articles written by colleagues that you can read if you'd like to pm me with an e-mail address. they are rather technical but they are pretty solid. i'll put a few of the abstracts up now:

Fetal development is a period sensitive to environmental influences such as maternal drug use. The most commonly used illicit drug by pregnant women is marijuana. The present study investigated the effects of in utero marijuana exposure on expression levels of opioid-related genes in the human fetal forebrain in light of the strong interaction between the cannabinoid and opioid systems. The study group consisted of 42 midgestation fetuses from saline-induced voluntary abortions. The opioid peptide precursors (preprodynorphin and preproenkephalin (PENK)) and receptor (mu, kappa and delta) mRNA expression were assessed in distinct brain regions. The effect of prenatal cannabis exposure was analyzed by multiple regression controlling for confounding variables (maternal alcohol and cigarette use, fetal age, sex, growth measure and post-mortem interval). Prenatal cannabis exposure was significantly associated with increased mu receptor expression in the amygdala, reduced kappa receptor mRNA in mediodorsal thalamic nucleus and reduced preproenkephalin expression in the caudal putamen. Prenatal alcohol exposure primarily influenced the kappa receptor mRNA with reduced levels in the amygdala, claustrum, putamen and insula cortex. No significant effect of prenatal nicotine exposure could be discerned in the present study group. These results indicate that maternal cannabis and alcohol exposure during pregnancy differentially impair opioid-related genes in distinct brain circuits that may have long-term effects on cognitive and emotional behaviors.

here's another:

The endocannabinoid system, including its receptors (CB1 and CB2), endogenous ligands(‘endocannabinoids’), synthesising and degrading enzymes, as well as transporter molecules, has been detected from the earliest stages of embryonic development and throughout pre- and postnatal development. In addition, the endocannabinoids, notably 2-arachidonyl glycerol, are also present in maternal milk. During three distinct developmental stages (i.e. embryonic implantation, prenatal brain development and postnatal suckling), the endocannabinoid system appears to play an essential role for development and survival. Thus, during early pregnancy, successful embryonic passage through the oviduct and implantation into the uterus both require critical enzymatic control of optimal anandamide levels at the appropriate times and sites. During foetal life, the cannabinoid CB1 receptor plays a major role in brain development, regulating neural progenitor differentiation into neurones and glia and guiding axonal migration and synaptogenesis. Postnatally, CB1 receptor blockade interferes with the initiation of milk suckling in mouse pups, by inducing oral motor weakness, which exposes a critical role for CB1 receptors in the initiation of milk suckling by neonates, possibly by interfering with innervation of the tongue muscles. Manipulating the endocannabinoid system by pre- and ⁄ or postnatal administration of cannabinoids or maternal marijuana consumption, has significant, yet subtle effects on the offspring. Thus, alterations in the dopamine, GABA and endocannabioid systems have been reported while enhanced drug seeking behaviour and impaired executive (prefrontal cortical) function have also been observed. The relatively mild nature of the disruptive effects of prenatal cannabinoids may be understood in the framework of the intricate timing requirements and frequently biphasic effects of the (endo)cannabinoids. In conclusion, the endocannabinoid system plays several key roles in pre- and postnatal development. Future studies should further clarify the mechanisms involved and provide a better understanding of the adverse effects of prenatal exposure, in order to design strategies for the treatment of conditions such as infertility, mental retardation and failure-to-thrive.

you can also just use common sense. during pregnancy the fetus is 100% linked to the mother. anything that goes into the mother is filtered through the ambiotic fluid and into the child. smoking marijuana puts carcinogens into the body... and thus into the baby. hope that helps. :peace:
you said smoking. what about if it is eaten?... made into cannabutter.
 

cackpircings

Well-Known Member

I see what you are saying “stopcallingmedude” but there are exceptions to the rule. Not only that you have to consider if the research in bias or not. I’m not a doctor so I can’t spit a bunch of fancy words out, but I can say that there is too many environmental factors that you have to account for when doing a study that internally affects a child inside the womb. Such as what did the woman eat while pregnant and using marijuana? Much like mixing a bunch of chemicals together to form new chemicals you need to have the whole picture and not the one variable. You claim to be a doc, so you have better credentials than I do, but I do have an education and an open mind. I have always been taught to pick things apart to get through the bull shit to the real evidence. All I am saying is did the test subjects all eat the same food; all go to bed at the same time. All have the same medical background…. So on and so on. I speak from experience (not to be confused with expertise) that my kid is 110% unaffected by the smoking.
 

Sunnysideup

Well-Known Member
I just don't like reading a bunch of medical terminology without references to back it up....It is just a bunch of words to me until I know who wrote it and what their credentials are....
 

cackpircings

Well-Known Member
I just don't like reading a bunch of medical terminology without references to back it up....It is just a bunch of words to me until I know who wrote it and what their credentials are....

And it’s still a bunch of words even with sources. The subjects are regulated to the control subject on any research project. I don’t see anything about that. Sounds like a bunch of bias (resrched by the tobacco industry) bull shiz.
 

Sunnysideup

Well-Known Member
I would like to see where he pulled these references....The SOURCE. Then that will allow me to make my own opinion with all the facts presented. I will wait and see where he got it from, and then I will make an educated decision. Without that info. it is useless.
 

cph

Well-Known Member
The thing that got me was the first one talked about doing the test on "42 midgestation fetuses from saline-induced voluntary abortions". Thats not inutero..:peace:
 

cackpircings

Well-Known Member
I don’t see why it is a big deal, just don’t over use the MJ while prago. Make sure you have good shiz, and just a couple of hits in the morning.
 

stopcallingmedude

Well-Known Member
I just don't like reading a bunch of medical terminology without references to back it up....It is just a bunch of words to me until I know who wrote it and what their credentials are....
And it’s still a bunch of words even with sources. The subjects are regulated to the control subject on any research project. I don’t see anything about that. Sounds like a bunch of bias (resrched by the tobacco industry) bull shiz.
i respect anyone who wants to take a closer look at anyone of these articles. that's great. read the articles and make your own judgements.

1st abstract: Journal of Neuroendocrinology; May 2008 Supplement, Vol. 20, p75-81, 7p, 1 chart, 2 graphs

2nd abstract: Pharmacogenomics Journal; 2006, Vol. 6 Issue 4, p255-264, 10p, 3 charts, 3 graphs

those are both cutting edge medical journals that are not "biases researched by the tobacco industry". they are peer reviewed by the top medical professionals in the world. check out the journal and read up on the subject. its fascinating.
 

Sunnysideup

Well-Known Member
i respect anyone who wants to take a closer look at anyone of these articles. that's great. read the articles and make your own judgements.

1st abstract: Journal of Neuroendocrinology; May 2008 Supplement, Vol. 20, p75-81, 7p, 1 chart, 2 graphs

2nd abstract: Pharmacogenomics Journal; 2006, Vol. 6 Issue 4, p255-264, 10p, 3 charts, 3 graphs

those are both cutting edge medical journals that are not "biases researched by the tobacco industry". they are peer reviewed by the top medical professionals in the world. check out the journal and read up on the subject. its fascinating.
I am on both of the pages now, thanks.:peace:
 
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