While cbd doesnt get you high and has medical implications thc's antitumor properties should not be ignored. Cb1 receptors are a very important aspect.
They should be used in combination, the tyoe of cancer can alter route of administration as well as what cannabinoids to take.
Specifically in this case a 1:1 or 2:1 ratio of thc:cbd is advised.
For example...
Antitumor Effects
During this 2-year study, groups of mice and rats were given various
doses of THC by
gavage. A dose-related decrease in the
incidence of
hepatic adenoma tumors and
hepatocellular carcinoma (HCC) was observed in the mice. Decreased incidences of
benign tumors (
polypsand adenomas) in other
organs(
mammary gland,
uterus, pituitary,
testis, and
pancreas) were also noted in the rats. In another study, delta-9-THC, delta-8-THC, and cannabinol (not cbd but cbn, very different) were found to inhibit the growth of Lewis lung adenocarcinom
a cells in vitro and
in vivo .
these compounds have been shown to induce
apoptosis in
glioma cells in
culture and induce
regression of glioma tumors in mice and rats, while they protect normal glial cells of astroglial and oligodendroglial lineages from apoptosis mediated by the CB1
receptor.
effects of delta-9-THC and a
synthetic agonist of the CB2 receptor were investigated in HCC (cbd is an antagonist or inverse agonist , thc actually hits cb1 and cb2 relatively equally, thcv is a strong cb2 agonist in comparison ).Both agents reduced the viability of HCC cells
in vitro and demonstrated antitumor effects in HCC subcutaneous xenografts
a subsequent study, the investigators found that the antiproliferative effect of CBD (also effects cb1) was counteracted by selective CB1 but not CB2 receptor antagonists, suggesting an involvement of CB1 receptors.
Etc
As for rso, I HIGHLY recommend not using such a product
