Black Folks and Aids

Nutes and Nugs

Well-Known Member
Africans are more susceptible to the AIDS virus. This is not really emerging news, it has been around for at least four or five years.

Genes, science, reality. Try some.

http://www.thebody.com/content/art21429.html
White women carry the heterozygous form of delta-32 far more frequently than any other women. About 1.2 percent of all white women who are HIV-negative carry the mutant gene. Only 0.6 percent of HIV-infected white women carry the gene. However, only 0.2 percent of HIV-negative African-American women carry the delta-32 gene. Among those who are HIV-positive, the frequency of delta-32 is only 0.1 percent.

"This supports mathematical predictions that having a low frequency of delta-32 in non-Caucasians may render those populations vulnerable to far greater and more explicit HIV epidemics," Burger said in a presentation on Wednesday at the Ninth Conference on Retroviruses and Opportunistic Infections. "And it may explain, in part, the rapid epidemics we see unfolding in Asia and Africa."
Thank You DesertDude!
That would be a logical explanation.
 
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UncleBuck

Well-Known Member

Nutes and Nugs

Well-Known Member
so you're in the clear to go fuck an AIDS ridden whore without a rubber (probably about the best you can hope to buy).

as you said, vagina is indeed preferable to man anus!
I have no intention of fucking whores nor a man's anus.

DesertDude gave the best answer so far as to why black folk are 8 times
more likely to contract hiv/aids.
 

earnest_voice

Well-Known Member
The paper states it's limited to 1% of Caucasian people and even then two copies of CCR5-delta32 gene is rare.

Paper also states;

It's not an airtight guarantee of never getting AIDS. Some unusual types of HIV can use other proteins for entering cells. Rarely, there have been people who have two mutant CCR5 genes who have died from AIDS.

Also, CCR5 is not the whole story of immunity to HIV infection. Some resistant people have been found who have two perfectly normal copies of CCR5. So other genes also contribute to slowing down HIV infection, and scientists are busy trying to identify them.
I think this gene is of little relevance to prevention of HIV transmission considering it's found in so few people as a % of total populace. The CDC link you posted in the OP lists some interesting facts in regards to prevalence and prevention;

Lack of awareness of HIV status can affect HIV rates in communities. Almost 85,000 HIV-infected people in the African American community in 2010 were unaware of their HIV status*. Diagnosis late in the course of HIV infection is common, which results in missed opportunities to get early medical care and prevent transmission to others.

The poverty rate is higher among African Americans than other racial/ethnic groups. The socioeconomic issues associated with poverty—including limited access to high-quality health care, housing, and HIV prevention education—directly and indirectly increase the risk for HIV infection, and affect the health of people living with and at risk for HIV. These factors may explain why African Americans have worse outcomes on the HIV continuum of care, including lower rates of linkage to care, retention in care, being prescribed HIV treatment, and viral suppression. New data from 2010 indicate that 75% of HIV-infected African Americans aged 13 or older are linked to care, 48% are retained in care, 46% are prescribed antiretroviral therapy, and only 35% are virally suppressed.

Stigma, fear, discrimination, homophobia, and negative perceptions about HIV testing can also place too many African Americans at higher risk. Many at risk for HIV fear discrimination and rejection more than infection and may choose not to seek testing.
*The highlighted portion should be of serious concern. 85,000 is alarming and these could most certainly lead to new cases.
 

UncleBuck

Well-Known Member
I have no intention of fucking whores nor a man's anus.

DesertDude gave the best answer so far as to why black folk are 8 times
more likely to contract hiv/aids.
you continue to get your information from a white supremacist and tell me how that works out for ya.
 

Nutes and Nugs

Well-Known Member
you continue to get your information from a white supremacist and tell me how that works out for ya.
His reply made the most sense though DesertDude owes us a citation to that info.

I really can't believe your "socio-economic status" theory.
If you really believe blacks are stupid, trodden down, sex and drug addicts you are a stupid man.
Er woman.
Whatever the fuck you are today.
 

desert dude

Well-Known Member
How could that be the logical explanation? Viral load is the main factor in transmission not race.
Google it. People who carry the particular homozygous gene pair are immune to HIV. This genotype is confined to northern Europeans. This is not to say that all caucasians are immune to HIV. HIV rates among Caucasians is lower than it is amongst Asians and Africans and this is the likely explanation. This is also not meant to discount all the other stuff, such as viral load and unsafe sexual practices.

This is not racist, and it is not controversial except among those whose politics dictate their acceptance of science.
 

desert dude

Well-Known Member
I can understand that you (the reader of this post) might be ignorant of the science behind HIV and the delta 32 gene configuration. After all, it is kind of complicated and most people are simply not interested in the subject. My following comments are not directed at you.

Quit with your racist bullshit. There is a pretty damn clear explanation why HIV transmission rates are significantly lower amongst Caucasians. The information about this is easily available, so educate yourself about HIV then go find some other "racist outrage" about which to be outraged.

A google search, "delta 32 HIV", yielded 3,620,000 hits. I suggest you progs read the first three, or have a conservative read them to you.

http://www.nature.com/scitable/blog/viruses101/hiv_resistant_mutation
http://www.sciencedaily.com/releases/2005/03/050325234239.htm
http://www.ncbi.nlm.nih.gov/pubmed/15815693

http://en.wikipedia.org/wiki/CCR5
At least one copy of CCR5-Δ32 is found in about 4–16% of people of European descent. It has been speculated that this allele was favored by natural selection during the Black Death for Northern Europeans, but further research has revealed that the gene did not protect against the Black Death.[19] The current hypothesis is of protection vs smallpox throughout Europe,[19] especially in the major trade cities and in isolated islands and archipelagos, such as Iceland and the Azores.[20]



http://genetics.thetech.org/original_news/news13
HIV can only enter certain cells. How does it find the right cells? By special proteins called receptors.


Receptors sit on the outside of cells to receive messages and transmit them into the cell. HIV grabs onto cells that have a receptor called CD4.


Cells with the CD4 receptor are an important part of the body's system for fighting all diseases (our immune system). HIV gradually destroys these cells and cripples the immune system.


It turns out that CD4 isn't enough. Another protein called CCR5 is needed as well. CCR5, called a co-receptor because it works with CD4, is the door that opens to allow HIV to enter the cell.


Many people who are resistant to HIV have a mutation in the CCR5 gene called CCR5-delta32. The CCR5-delta32 mutation results in a smaller protein that isn't on the outside of the cell anymore. Most forms of HIV cannot infect cells if there is no CCR5 on the surface.


People with two copies of the CCR5 delta32 gene (inherited from both parents) are virtually immune to HIV infection. This occurs in about 1% of Caucasian people.


One copy of CCR5-delta32 seems to give some protection against infection, and makes the disease less severe if infection occurs. This is more common, it is found in up to 20% of Caucasians.

Should everyone be tested for this mutation? Not necessarily. It would be dangerous to assume you are completely safe from infection if you have the CCR5-delta32 mutation.

It's not an airtight guarantee of never getting AIDS. Some unusual types of HIV can use other proteins for entering cells. Rarely, there have been people who have two mutant CCR5 genes who have died from AIDS.

Also, CCR5 is not the whole story of immunity to HIV infection. Some resistant people have been found who have two perfectly normal copies of CCR5. So other genes also contribute to slowing down HIV infection, and scientists are busy trying to identify them.
 

DonAlejandroVega

Well-Known Member
I can understand that you (the reader of this post) might be ignorant of the science behind HIV and the delta 32 gene configuration. After all, it is kind of complicated and most people are simply not interested in the subject. My following comments are not directed at you.

Quit with your racist bullshit. There is a pretty damn clear explanation why HIV transmission rates are significantly lower amongst Caucasians. The information about this is easily available, so educate yourself about HIV then go find some other "racist outrage" about which to be outraged.

A google search, "delta 32 HIV", yielded 3,620,000 hits. I suggest you progs read the first three, or have a conservative read them to you.

http://www.nature.com/scitable/blog/viruses101/hiv_resistant_mutation
http://www.sciencedaily.com/releases/2005/03/050325234239.htm
http://www.ncbi.nlm.nih.gov/pubmed/15815693

http://en.wikipedia.org/wiki/CCR5
At least one copy of CCR5-Δ32 is found in about 4–16% of people of European descent. It has been speculated that this allele was favored by natural selection during the Black Death for Northern Europeans, but further research has revealed that the gene did not protect against the Black Death.[19] The current hypothesis is of protection vs smallpox throughout Europe,[19] especially in the major trade cities and in isolated islands and archipelagos, such as Iceland and the Azores.[20]



http://genetics.thetech.org/original_news/news13
HIV can only enter certain cells. How does it find the right cells? By special proteins called receptors.

Receptors sit on the outside of cells to receive messages and transmit them into the cell. HIV grabs onto cells that have a receptor called CD4.

Cells with the CD4 receptor are an important part of the body's system for fighting all diseases (our immune system). HIV gradually destroys these cells and cripples the immune system.

It turns out that CD4 isn't enough. Another protein called CCR5 is needed as well. CCR5, called a co-receptor because it works with CD4, is the door that opens to allow HIV to enter the cell.

Many people who are resistant to HIV have a mutation in the CCR5 gene called CCR5-delta32. The CCR5-delta32 mutation results in a smaller protein that isn't on the outside of the cell anymore. Most forms of HIV cannot infect cells if there is no CCR5 on the surface.

People with two copies of the CCR5 delta32 gene (inherited from both parents) are virtually immune to HIV infection. This occurs in about 1% of Caucasian people.


One copy of CCR5-delta32 seems to give some protection against infection, and makes the disease less severe if infection occurs. This is more common, it is found in up to 20% of Caucasians.

Should everyone be tested for this mutation? Not necessarily. It would be dangerous to assume you are completely safe from infection if you have the CCR5-delta32 mutation.

It's not an airtight guarantee of never getting AIDS. Some unusual types of HIV can use other proteins for entering cells. Rarely, there have been people who have two mutant CCR5 genes who have died from AIDS.

Also, CCR5 is not the whole story of immunity to HIV infection. Some resistant people have been found who have two perfectly normal copies of CCR5. So other genes also contribute to slowing down HIV infection, and scientists are busy trying to identify them.

a "superior" gene......for sure :)
 
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